Gain, Not Noise
When input from the inner ear drops — through noise damage, age or circulation — the auditory cortex compensates by amplifying what remains. Tinnitus is often that amplification, which is why it is louder in silence and at night.
This also explains why stress reliably worsens it: the same networks that set auditory gain are modulated by the stress response.
Circulation and Cervical Input
The inner ear has an unusually demanding blood supply and no collateral circulation. Blood pressure, blood sugar, anaemia and vascular stiffness all show up there early.
Jaw and upper cervical mechanics matter too. Tinnitus that changes when you clench, chew or turn your head is pointing at TMJ or cervical input rather than at the cochlea alone.
Metabolic and Medication Drivers
Insulin resistance, low ferritin, low B12, thyroid dysfunction and magnesium depletion all appear repeatedly in tinnitus histories. So do medications: high-dose NSAIDs, some antibiotics, loop diuretics and certain chemotherapy agents.
These are checkable, and some are reversible, which is why a metabolic workup belongs in the assessment rather than only an audiogram.
